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Nutrients Safely Boost Metabolism in Rats
July 5, 2008
Nutrients can activate gene metabolism within cells, not only improving the metabolism of glucose and fatty acids but doing so without any adverse side effects.
In the study researchers used R-Alpha Lipoic Acid (RLA) and Acetyl-L-Carnitine (ALC)1 as the primary nutrients tested, along with niacin and biotin (two B vitamins typically found in a multiple vitamin or B Complex) as cofactors to enhance the use of RLA and ALC. They found these nutrients dramatically improved the metabolism of sugar and fatty acids by directly activating genes in the mitochondria (your cell's car engines), even increasing the horsepower of these cell engines so that more calories could be burned more efficiently – changing the source of the problem without any adverse side effects.
When doctors try to lower blood sugar with drugs they often fail to ask “Where is the sugar going?” If it is being transferred to fat, i.e. weight gain, it is clearly going in the wrong direction. If it is being transferred into the mitochondria of cells to be burned as fuel then health is more likely to occur. Drugs are a very poor choice for this activity, and attempts to improve metabolism with drugs come at a high price of damaging side effects.
Exercise is known to condition your mitochondria back to health. Strength training builds muscle over time – the more muscle you have the more mitochondria you possess. When you do aerobics and become more fit you condition your mitochondria to work better. On top of that you can add nutrients that improve mitochondrial function. The current study relies on RLA and ALC to prove this point – two great nutrients with numerous metabolic and health benefits. There are many other options that are synergistic with this concept – including nutrients for leptin, thyroid, and adrenal support. The main underlying point is that when you improve the function of mitochondria you improve metabolism of both blood sugar and fatty acid. Nutrients stand alone as the safe and effective way to support consistent exercise to accomplish this goal.
In the study researchers used R-Alpha Lipoic Acid (RLA) and Acetyl-L-Carnitine (ALC)1 as the primary nutrients tested, along with niacin and biotin (two B vitamins typically found in a multiple vitamin or B Complex) as cofactors to enhance the use of RLA and ALC. They found these nutrients dramatically improved the metabolism of sugar and fatty acids by directly activating genes in the mitochondria (your cell's car engines), even increasing the horsepower of these cell engines so that more calories could be burned more efficiently – changing the source of the problem without any adverse side effects.
When doctors try to lower blood sugar with drugs they often fail to ask “Where is the sugar going?” If it is being transferred to fat, i.e. weight gain, it is clearly going in the wrong direction. If it is being transferred into the mitochondria of cells to be burned as fuel then health is more likely to occur. Drugs are a very poor choice for this activity, and attempts to improve metabolism with drugs come at a high price of damaging side effects.
Exercise is known to condition your mitochondria back to health. Strength training builds muscle over time – the more muscle you have the more mitochondria you possess. When you do aerobics and become more fit you condition your mitochondria to work better. On top of that you can add nutrients that improve mitochondrial function. The current study relies on RLA and ALC to prove this point – two great nutrients with numerous metabolic and health benefits. There are many other options that are synergistic with this concept – including nutrients for leptin, thyroid, and adrenal support. The main underlying point is that when you improve the function of mitochondria you improve metabolism of both blood sugar and fatty acid. Nutrients stand alone as the safe and effective way to support consistent exercise to accomplish this goal.
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